Fluid and Electrolytes PDF

Title Fluid and Electrolytes
Author Abdirahman Noah
Course Adult Health
Institution Edith Cowan University
Pages 23
File Size 1.1 MB
File Type PDF
Total Downloads 4
Total Views 136

Summary

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Description

Fluid and Electrolytes: Balance and Disturbance Fluid and Electrolyte Balance •

Necessary for life, homeostasis



Nursing role: help prevent, treat fluid, electrolyte disturbances

Fluid •

Approximately 60% of typical adult is fluid – Varies with age, body size, gender



Intracellular fluid



Extracellular fluid – Intravascular: the fluid within the blood vessels e.g. plazma – Interstitial: fluid surrounds the cell (e.g. lymph – Transcellular: cerebrospinal, pericardial, synovial, intraocular, pleural, sweat and digestive secretion



“Third spacing”: loss of ECF into space that does not contribute to equilibrium

Third spacing  Third spaces are extra-cellular body spaces where fluid can accumulate  This accumulated fluid is useless to the body  Unavailable for use as reserve fluid  Unable to transport nutrients  Common locations for third space fluid to accumulate  Tissue spaces (edema)  Abdomen (ascites)  Pleural spaces (pleural effusion)  Pericardial space (pericardial effusion)  Electrolytes •

Active chemicals that carry positive (cations), negative (anions) electrical charges – Major cations: sodium, potassium, calcium, magnesium, hydrogen ions – Major anions: chloride, bicarbonate, phosphate, sulfate, and proteinate ions



Electrolyte concentrations differ in fluid compartments

Regulation of Fluid

Regulation of Fluid •

Osmosis: area of low solute concentration to area of high solute concentration



Diffusion: solutes move from area of higher concentration to one of lower concentration



Filtration: movement of water, solutes occurs from area of high hydrostatic pressure to area of low hydrostatic pressure



Active transport: physiologic pump that moves fluid from area of lower concentration of one of higher concentration

Active Transport •

Physiologic pump that moves fluid from area of lower concentration to one of higher concentration



Movement against concentration gradient



Sodium-potassium pump: maintains higher concentration of extracellular sodium, intracellular potassium



Requires adenosine (ATP) for energy

Question •

Tell whether the following statement is true or false:



Osmosis is the movement of a substance from an area of higher concentration to one of lower concentration.

Answer •

False.



Rationale: Diffusion is the movement of a substance from an area of higher concentration to one of lower concentration. The concentration of dissolved substances draws fluid in that direction. Osmosis is the movement of fluid, through a semipermeable membrane, from an area of low solute concentration to an area of high solute concentration until the solutions are of equal concentration.

Routes of Gains and Losses •

Gain – Dietary intake of fluid, food or enteral feeding Ingested fluid (60%) and solid food (30%) – Metabolic water or water of oxidation (10%) – Parenteral fluids



Loss – Kidney: urine output – Skin loss: sensible, insensible losses – Lungs – GI tract – Other – Urine (60%) and feces (4%) – Insensible losses (28%), sweat (8%)

Water Intake and Output

Question •

What is the average daily urinary output in an adult?



0.5 L



1.0 L



1.5 L



2.5 L

Answer •

C. 1.5 L



Rationale: Vital to the regulation of fluid and electrolyte balance, the kidneys normal filter 170 L of plasma every day in the adult, while excreting only 1.5 L of urine.



Regulation of Water Intake The hypothalamic thirst center is stimulated: – By a decline in plasma volume of 10%–15% – By increases in plasma osmolality of 1–2% – Via baroreceptor input, angiotensin II, and other stimuli



Thirst is reduced as soon as we begin to drink water



Feedback signals that inhibit the thirst centers include: – Moistening of the mucosa of the mouth and throat

– Activation of stomach and intestinal stretch receptors Regulation of Water Output •

Obligatory water losses include: – Insensible water losses from lungs and skin – Water that accompanies undigested food residues in feces



Obligatory water loss reflects the fact that: – Kidneys excrete 900-1200 mOsm of solutes to maintain blood homeostasis – Urine solutes must be flushed out of the body in water

Influence and Regulation of ADH •

Water reabsorption in collecting ducts is proportional to ADH release



Low ADH levels produce dilute urine and reduced volume of body fluids



High ADH levels produce concentrated urine



Hypothalamic osmoreceptors trigger or inhibit ADH release



Factors that specifically trigger ADH release include prolonged fever; excessive sweating, vomiting, or diarrhea; severe blood loss; and traumatic burns

Mechanisms and Consequences of ADH Release

Fluid Volume Imbalances •

Fluid volume deficit (FVD): hypovolemia



Fluid volume excess (FVE): hypervolemia

Fluid Volume Deficit •

Loss of extracellular fluid exceeds intake ratio of water – Electrolytes lost in same proportion as they exist in normal body fluids



Dehydration: loss of water along with increased serum sodium level – May occur in combination with other imbalances



Dehydration – Causes: fluid loss from vomiting, diarrhea, GI suctioning, sweating, decreased intake, inability to gain access to fluid – Risk factors: diabetes insipidus, adrenal insufficiency, osmotic diuresis, hemorrhage, coma,



Manifestations: rapid weight loss, decreased skin turgor, oliguria, concentrated urine, postural hypotension, rapid weak pulse, increased temperature, cool clammy skin due to vasoconstriction, lethargy, thirst, nausea, muscle weakness, cramps



Laboratory data: elevated BUN in relation to serum creatinine, increased hematocrit

– Serum electrolyte changes may occur •

Medical management: provide fluids to meet body needs – Oral fluids – IV solutions (isotonic electrolytes solution; e.g. ringer lactate solution or 0.9 sodium chloride)

Fluid Volume Deficit - Nursing Management •

I&O, VS



Monitor for symptoms: skin and tongue turgor, mucosa, UO, mental status



Measures to minimize fluid loss



Oral care



Administration of oral fluids



Administration of parenteral fluids

Question •

What is a major indicator of extracellular FVD?



Full and bounding pulse



Drop in postural blood pressure



Elevated temperature



Pitting edema of lower extremities

Answer •

B. Drop in postural blood pressure



Rationale: FVD signs and symptoms include acute weight loss; decreased skin turgor; oliguria; concentrated urine; orthostatic hypotension due to volume depletion; a weak, rapid heart rate; flattened neck veins; increased temperature; thirst; decreased or delayed capillary refill; decreased central venous pressure; cool, clammy, pale skin related to peripheral vasoconstriction; anorexia; nausea; lassitude; muscle weakness; and cramps. Clinical manifestations of FVE result from expansion of the ECF and include edema, distended neck veins, and crackles (abnormal lung sounds).

Fluid Volume Excess •

Due to fluid overload or diminished homeostatic mechanisms



Risk factors: heart failure, renal failure, cirrhosis of liver



Contributing factors: excessive dietary sodium or sodium-containing IV solutions



Manifestations: edema, distended neck veins, abnormal lung sounds (crackles), tachycardia, increased BP, pulse pressure and CVP, increased weight, increased UO, shortness of breath and wheezing



Medical management: directed at cause, restriction of fluids and sodium, administration of diuretics

Edema: is defined as a palpable swelling caused by a increase in interstitial fluid volume Edema, other than localized edema, does not become clinically apparent until the interstitial volume has increased by 2.5 to 3 liters Disorders of Water Balance: Edema •

Atypical accumulation of fluid in the interstitial space, leading to tissue swelling



Caused by anything that increases flow of fluids out of the bloodstream or hinders their return



Factors that accelerate fluid loss include: – Increased blood pressure, capillary permeability – Incompetent venous valves, localized blood vessel blockage – Congestive heart failure, hypertension, high blood volume

Edema •

Hindered fluid return usually reflects an imbalance in colloid osmotic pressures



Hypoproteinemia – low levels of plasma proteins – Forces fluids out of capillary beds at the arterial ends – Fluids fail to return at the venous ends – Results from protein malnutrition, liver disease, or glomerulonephritis



Blocked (or surgically removed) lymph vessels: – Cause leaked proteins to accumulate in interstitial fluid – Exert increasing colloid osmotic pressure, which draws fluid from the blood



Interstitial fluid accumulation results in low blood pressure and severely impaired circulation

Fluid Volume Excess - Nursing Management •

I&O and daily weights; assess lung sounds, edema, other symptoms; monitor responses to medications- diuretics



Promote adherence to fluid restrictions, patient teaching related to sodium and fluid restrictions



Monitor, avoid sources of excessive sodium, including medications



Promote rest



Semi-Fowler’s position for orthopnea



Skin care, positioning/turning

Electrolyte Imbalances •

Sodium (Na= 135-145) : hyponatremia, hypernatremia



Potassium (k= 3.5-5.0) : hypokalemia, hyperkalemia



Calcium (Ca= 9-11): hypocalcemia, hypercalcemia



Magnesium (Mg= 1.8-3): hypomagnesemia, hypermagnesemia



Phosphorus (P=3.0-4.5): hypophosphatemia, hyperphosphatemia



Chloride (Cl= 96-106): hypochloremia, hyperchloremia

Hyponatremia •

Serum sodium less than 135 mEq/L



Causes:



 Adrenal insufficiency,  Water intoxication,  SIADH (syndrome of inappropriate adh secretion) or  Losses by vomiting, diarrhea, sweating, diuretics Manifestations:



 Poor skin turgor,  Dry mucosa,  Headache,  Decreased salivation,  Decreased bp,  Nausea,  Abdominal cramping,  Neurologic changes Medical management:



 Water restriction,  Sodium replacement Nursing management:  Assessment and prevention, dietary sodium and fluid intake,  Identify and monitor at-risk patients, effects of medications (diuretics, lithium)

Hypernatremia •

Serum sodium greater than 145mEq/L



Causes:



 Excess water loss,  Excess sodium administration,  Diabetes insipidus,  Heat stroke,  Hypertonic IV solutions Manifestations:



 Thirst;  Elevated temperature;  Dry, swollen tongue;  Sticky mucosa;  Neurologic symptoms; restlessness; weakness Note: thirst may be impaired in elderly or the ill



Medical management:



 Hypotonic electrolyte solution or D5W Nursing management:     

Assessment and prevention, Assess for OTC (Over the counter drugs) Sources of sodium, Offer and encourage fluids to meet patient needs, Provide sufficient water with tube feedings

Hypokalemia •

Below-normal serum potassium (...


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