Title | Fluid & Electrolytes ATI |
---|---|
Author | Tommy Garrett |
Course | Introducing Nutrition |
Institution | University of California Davis |
Pages | 7 |
File Size | 195.3 KB |
File Type | |
Total Downloads | 59 |
Total Views | 134 |
Fluids...
ATI 57 & 58 Chapter 57: Fluid Imbalance Fluid Volume Deficit/ Hypovolemia EX: Hypovolemia & Dehydration
Risk Factors: GI loss- vomiting, NG suction, diarrhea Diaphoresis Diuretics, diabetes insipidus, CKD Peritonitis, intestinal obstruction, ascites, burns Excessive loss from wound Hemorrhage Altered intake Dehydration: Hyperventilation Prolonged fever DKA Enteral feeding w/o water *Older adults @ risk-> decrease in ability to detect thirst Expected Findings: VS: Hypothermia Tachycardia Thready pulse Hypotension Orthostatic Hypotension Decrease CVP Tachypnea Hypoxia Others: Dizziness, syncope, confusion, weakness, fatigue Thirst, dry mm, dry tongue, n/v/anorexia, weight loss Oliguria Diminished cap refill, cool clammy skin, diaphoresis, flattened neck veins, decreased skin turgor Older adults* skin turgor not as reliable bc loss of elasticity
Fluid Volume Excess/ Hypervolemia EX: Hypervolemia & Overhydration *Can lead to pulmonary edema and heart failure* Risk Factors: Kidney failure Age related changes in cardiovascular and kidney function Excessive sodium intake from IV fluids, diet, or medications Overhydration: Water replacement without electrolyte replacement Syndrome of inappropriate antidiuretic hormone (SIADH) (excess ADH) Head injuries Barbiturates Anesthetics Expected Findings: VS: Tachycardia Bounding Pulse Hypertension Increased CVP Tachypnea Others: Confusion Muscle weakness Weight gain Ascites Dyspnea Orthopnea Crackles Edema Distended neck veins
Labs: Increase serum osmolarity Increased Urine specific gravity: > 1.030 Increased sodium: >145
Labs: Decreased serum osmolarity: hypotension Check neurological status to determine level of consciousness Assess heart rhythm Isotonic Infusions: 0.9% NS, LR, D5W I&O; encourage fluids Alert if Cell shrinks; hypertonicity Complications: coma, seizures, resp arrest Risk Factors: Risk Factors: Deficient ECF volume Water deprivation (NPO)
GI loss: vomiting, NG suctioning, diarrhea, tap water enemas Renal losses: diuretics, kidney disease, adrenal insufficiency excessive sweating Skin losses: burns, wound drainage, edema Excessive oral water intake, SIADH Heart failure, cirrhosis, nephrotic syndrome Excessive hypotonic fluids Inadequate sodium intake (NPO status) Hyperglycemia Age related : older adults, use of diuretics Findings: Hypothermia Tachycardia Rapid thread pulse Hypotension Orthostatic hypotension Headache Confusion Lethargy Muscle weakness Fatigue Decreased DTRs Seizures Coma Hyperactive bowel sounds Abdominal cramping n/v/a
Labs: < 135 Sodium Decreased serum osmolarity < 280 Decreased Urine specific gravity: altered thirst mechanism r/t altered thirst mechanism
Findings: Hyperthermia Tachycardia Orthostatic hypotension Restlessness Disorientation Irritability Muscle twitching Muscle weakness Seizures Decreased LOC Reduced to absent DTR Thirst, dry mm Dry and swollen tongue Hyperactive bowel sounds Abdominal cramping, nausea Edema Oliguria Warm flushed skin Labs: >145 Sodium Increased osmolarity > 295 Increased USG: > 1.030 Patient Care: LOC and ensure safety
Oral hygiene to decrease thirst
Fluid restrictions prescribed Monitor respiratory status if muscle weakness is present Restrict water intake if fluid overload Hypertonic IV therapy: >0.9 NS
I&O Hypotonic IV fluids: 5.0 Risk Factors: Risk Factors: IV potassium admin, salt substitutes, Hyperaldosteronism Vomiting, ng suctioning, diarrhea, blood transfusions excessive laxative Insufficient insulin, DKA Furosemide (diuretics that lose K) Uncontrolled diabetes Kidney failure, severe dehydration, K Diaphoresis, wound losses Metabolic alkalosis sparing diuretics, ACE inhibitors, Tissue repair of burns, trauma, adrenal insufficiency Older adult-> decreased kidney starvation TPN function Findings: Findings: Hyperthermia Slow, irregular pulse Weak irregular pulse Hypotension Hypotension Irritability, confusion, weakness w/ Orthostatic hypotension ascending flaccid paralysis Respiratory distress Paresthesia Bilateral muscle weakness w/ Lack of reflexes respiratory collapse and paralysis V. Fib, peaked T waves, widened QRS, cardiac arrest Muscle cramping Decreased muscle tone + hypoactive Diarrhea reflexes Abdominal cramps, hyperactive bowel sounds Mental confusion PVCs, bradycardia, blocks ventricular tachycardia, flattening T waves, and ST depression Hypoactive bowel sounds, abdominal distension, constipation, ileus, nausea, vomiting, anorexia Anxiety Labs: Labs: < 3.5 Potassium > 5.0 potassium
Metabolic alkalosis EKG needed Patient Care: Replace potassium Monitor for urine output Monitor breathing Monitor cardiac rhythm Monitor clients receiving digoxin bc of digoxin toxicity LOC IV potassium supplementation Dilute to 1mEq-10 mL and infuse slowly no faster than 10 mEq/hr Monitor for phlebitis Never IV bolus bc cardiac arrest risk
Metabolic acidosis EKG needed Patient Care: Decrease K intake: stop infusion of IV potassium hold PO potassium, K restricted diet IV fluids with dextrose and regular insulin Monitor cardiac rhythm Prepare for dialysis if needed Meds: Loop diuretics-> furosemide (if good kidney function) Sodium polystyrene sulfonate (Kayexalate)-> poop the K out
Calcium 9- 10.5 mg/dL o Function of cardiovascular, neuromuscular, endocrine, blood clotting, bone and teeth formation -> NEED Vit D to absorb the Calcium!! Hypocalcemia: < 9 Hypercalcemia: >10.5 Risk Factors: Risk Factors: Thiazide diuretics Chronic diarrhea Steatorrhea Increased calcium intake and absorption Malabsorption syndromes: Crohn’s Vit D Deficiency Hyperparathyroidism Rapid infusion of blood Bone cancer Post thyroidectomy Paget’s disease Hypoparathyroidism Chronic immobility Long term glucocorticoid use Hyperthyroidism Findings: Findings: Decreased reflexes Muscle Twitches/ Tetany Bone pain Numbness & Tingling around mouth Flank pain if renal calculi and fingers Frequent painful muscle spasms Dysrhythmias Hyperactive DTR Increased risk for blood clot n/v/a/constipation Positive Chvostek’s sign (tapping on facial nerve triggering the twitch) weakness, lethargy confusion, decreased LOC Positive Trousseau’s sign (hand/finger spasms with sustained blood pressure cuff inhalation) Laryngospasm
Cardiovascular: Weak, thread pulse Prolonged QT interval and ST segments Others: Diarrhea, abdominal cramping Seizures due to the overstim of CNS Labs: < 9 calcium Patient Care: Oral or IV calcium supplements-> monitor respiratory and cardiovascular status Initiate seizure precautions (padded beds, suctioning nearby, position to prevent aspiration, protect head) Keep emergency equipment on standby Encourage foods high in calcium: dairy, dark green vegetables
Labs: > 10.5 Patient Care: Increase the activity level
Magnesium: 1.3-2.1 mEq/L o Small amount in ECF Hypomagnesemia: 2.1 Risk Factors: CKD, AKI Adrenal insufficiency Laxatives or antacids
Findings: Diminished DTRs Muscle paralysis Shallow respirations, decreased respiratory rate Bradycardia, hypotension Dysrhythmias, cardiac arrest lethargy
Patient Care: Frequent focused assessments* Notify provider of changes or absent
Oral can cause diarrhea and increase MG depletion ; IV is best Monitor DTRs for hyper magnesia Whole grains and dark green vegetables Implement seizure precautions
reflexes Monitor resp and cardiac Admin loop diuretics and magnesium free IV fluids Administer calcium for severe cardiac changes...